CBD Oil for Nightmares: REM Sleep, PTSD, and the Evidence | PureCraft CBD
By PureCraft Editorial Team | PureCraft CBD | Updated October 2026
Nightmares are more than unpleasant dreams — they're a disruption of the brain's most critical emotional processing system. For the roughly 5–8% of adults who experience nightmares regularly, and for the far higher proportion of people with PTSD, chronic nightmare disorder can fragment sleep, worsen anxiety, and create a feedback loop that makes both the trauma and the sleep disorder harder to treat.
CBD oil has attracted growing interest as a potential tool for nightmare reduction, particularly among people with PTSD-associated nightmares. But the mechanisms here are genuinely nuanced — CBD doesn't simply "stop bad dreams." It works through several converging pathways: modulating the endocannabinoid system during REM sleep, reducing amygdala hyperreactivity, supporting fear extinction consolidation, and dampening the HPA axis response that fuels noradrenergic hyperactivity at night.
This guide covers the neuroscience of nightmares, the evidence base for CBD oil across nightmare types, dosing considerations, the important REM suppression trade-off at higher doses, and how CBD oil compares to established treatments like prazosin and Image Rehearsal Therapy. If you're exploring CBD for sleep support or PTSD specifically, this is one of the most mechanistically rich applications — and one where honest interpretation of the evidence really matters.
Types of Nightmares: Not All Bad Dreams Are the Same
Understanding how CBD oil might help requires distinguishing between nightmare types — because the underlying mechanisms differ, and so does the evidence for CBD's relevance.
Idiopathic Nightmares
These are distressing dreams with no identifiable trauma origin. They tend to be stress-related, often escalating during periods of elevated anxiety, poor sleep hygiene, or life disruption. Approximately 5–8% of adults experience these regularly. The content is typically threatening — being chased, falling, confronting danger — but doesn't replay a specific memory. CBD oil's anxiolytic and HPA axis-modulating effects are most relevant here, working through general stress reduction rather than trauma-specific mechanisms.
PTSD-Associated Nightmares (Trauma Nightmares)
These are the most clinically studied category in the context of cannabinoids. They involve the replay of traumatic events or thematically related content, are associated with a hyper-reactive amygdala during REM sleep, and are driven partly by noradrenergic hyperactivity originating from the locus coeruleus. They tend to cause fragmented sleep, contribute to hyperarousal, and can significantly worsen daytime PTSD symptoms. This is where CBD oil's evidence base — though still preliminary — is strongest and most mechanistically plausible. See our dedicated post on CBD oil for PTSD for the full picture.
Nightmare Disorder
This is the clinical diagnosis — repeated nightmares that cause significant distress or impairment in waking life, not better explained by another condition. It's distinct from occasional bad dreams. Treatment typically involves Image Rehearsal Therapy (IRT) as the gold standard, sometimes combined with pharmacotherapy. CBD oil may be relevant as an adjunct, but cannot replace evidence-based treatment for this diagnosis.
REM Sleep Behavior Disorder (RBD)
RBD involves physically acting out dreams — a loss of the normal muscle atonia that characterizes REM sleep. The mechanism is fundamentally different from nightmare disorder (it's a motor control issue, not an emotional processing dysregulation), and CBD oil is not established as a treatment for RBD. Anyone experiencing physical dream enactment should consult a neurologist, as RBD can be an early marker of neurodegenerative conditions.
The Neuroscience of Nightmares: REM Sleep, Fear Memory, and the Amygdala
How REM Sleep Normally Processes Fear — and What Goes Wrong in Nightmares
REM sleep is the brain's primary stage for emotional memory consolidation. During healthy REM cycles, the amygdala is highly active — processing emotional memories from the day — but the ventromedial prefrontal cortex (vmPFC) maintains inhibitory control over it. This balance allows emotional processing to occur without triggering a full threat response. Fear memories are replayed, contextualized, and gradually lose their emotional charge through a process called fear extinction consolidation.
In trauma nightmares, this system breaks down. The amygdala becomes hyperactivated — it's not just active, it's dysregulated — and the vmPFC loses its inhibitory grip. Fear memories replay without being extinguished. The locus coeruleus releases excess norepinephrine (NE), driving a physiological stress response during REM that mirrors the original trauma. Heart rate increases, breathing shallows, and the sleeper wakes — often abruptly, often in a state of high arousal.
This noradrenergic hyperactivity is why prazosin — an alpha-1 adrenergic receptor blocker — is the most evidence-supported pharmacological treatment for PTSD nightmares. By blocking NE activity at the alpha-1 receptor during sleep, prazosin reduces the physiological storm that drives trauma nightmares. It's a targeted, direct mechanism.
The endocannabinoid system (ECS) is deeply embedded in this process. CB1 receptors are concentrated in both the amygdala and the vmPFC. Endocannabinoids — particularly anandamide — facilitate fear extinction learning, which means the ECS plays a direct role in whether fear memories are resolved or replayed during REM. Anandamide facilitates extinction consolidation; 2-AG helps modulate noradrenergic tone. CBD's capacity to indirectly enhance endocannabinoid signaling makes it mechanistically relevant here — though through a less direct pathway than prazosin.
How CBD Oil May Affect Nightmares: Five Mechanisms
1. Anxiolytic Effects and Pre-Sleep Hyperarousal Reduction
Pre-sleep anxiety and hyperarousal are primary triggers for nightmares. CBD oil's well-documented anxiolytic effects — supported by multiple human trials and robust animal model data — reduce the physiological and psychological activation that a person carries into sleep. Less hyperarousal entering REM means a less reactive amygdala during REM. This is the most well-supported mechanism and the one most relevant to stress-related nightmares. For more on this pathway, see our guide on CBD oil for anxiety.
2. 5-HT1A Partial Agonism and Amygdala Reactivity
CBD acts as a partial agonist at serotonin 5-HT1A receptors. This receptor subtype is expressed throughout the amygdala and plays a significant role in modulating fear and anxiety responses. 5-HT1A agonism reduces amygdala firing in response to threat cues — a mechanism that may directly reduce the amygdala hyperactivation that characterizes trauma nightmares during REM. This is also likely part of why CBD's anxiolytic effects are relatively rapid compared to chronic SSRIs, which require weeks of downstream 5-HT1A desensitization.
3. ECS Enhancement and Fear Extinction Consolidation
CBD inhibits FAAH (fatty acid amide hydrolase), the enzyme that breaks down anandamide. By slowing anandamide degradation, CBD increases endocannabinoid tone. Because anandamide is required for fear extinction learning — and because CB1 receptor signaling in the amygdala and vmPFC is necessary for extinction memory consolidation — enhanced ECS activity during REM may support the brain's natural nightmare-resolution process. This is a compelling theoretical mechanism, though direct evidence in human nightmare studies remains limited.
4. HPA Axis Modulation and Reduced Nocturnal Cortisol
Elevated cortisol — driven by HPA axis hyperactivation in stress and PTSD — contributes to noradrenergic hyperactivity during sleep. CBD has demonstrated HPA axis modulating effects in multiple studies, reducing cortisol responses to stress. Lower cortisol going into sleep translates to less locus coeruleus activation during REM, less norepinephrine release, and a calmer amygdala environment — indirectly reducing the physiological substrate for trauma nightmares. This is also relevant for people experiencing chronic stress without a PTSD diagnosis.
5. REM Suppression at Higher Doses (With an Important Trade-Off)
At higher doses, CBD can suppress REM sleep — meaning fewer REM cycles and less total REM time. Fewer REM cycles means fewer opportunities for nightmares to occur. This sounds like a straightforward benefit, but it comes with a significant trade-off: REM sleep is essential for emotional memory processing, learning consolidation, and psychological resilience. Chronic REM suppression — even from a well-tolerated compound — can impair the brain's capacity to process difficult emotions and consolidate memories. For PTSD specifically, this is a meaningful concern: you may reduce nightmare frequency while simultaneously reducing the brain's capacity for fear extinction during REM. Lower to moderate doses are generally preferred for this reason.
CBD Oil Dose and REM: The Trade-Off Table
One of the most important — and underexplained — aspects of CBD for nightmares is the dose-dependent relationship with REM sleep. Higher doses don't simply produce more benefit.
| CBD Dose | REM Effect | Nightmare Impact | Trade-off |
|---|---|---|---|
| Low (15–25mg) | Minimal REM change | Indirect via anxiety reduction | Best preservation of emotional processing; may not reduce nightmares directly |
| Moderate (25–60mg) | Slight REM reduction | Reduced nightmare frequency | Balanced approach; generally recommended starting point for nightmare reduction |
| High (60–150mg+) | Significant REM suppression | Fewer nightmares | May impair emotional processing and memory consolidation; REM rebound on discontinuation |
For PTSD nightmares specifically, moderate doses are generally favored — enough to reduce nightmare intensity and frequency without substantially impairing the REM sleep that supports fear extinction. Very high doses that eliminate REM cycles may produce short-term relief but work against the brain's long-term trauma resolution capacity.
For more on finding your optimal dose, see our CBD oil dosage guide.
CBD Oil Relevance by Nightmare Type
| Nightmare Type | CBD Relevance | Primary Mechanism | Evidence Level |
|---|---|---|---|
| PTSD nightmares | HIGH | ECS/fear extinction, REM modulation, HPA axis | Moderate (mostly extrapolated) |
| Stress-related nightmares | MODERATE-HIGH | Anxiolytic, HPA modulation | Low-moderate |
| Idiopathic nightmares | MODERATE | Anxiolytic, sleep quality improvement | Low |
| Nightmare Disorder (clinical) | MODERATE | Adjunct; therapist involvement needed | Low |
| REM Sleep Behavior Disorder | LOW | Not mechanism-matched | Very low |
The Evidence Base: What Research Actually Shows
It's important to be direct about the state of the evidence: there are no large-scale, placebo-controlled RCTs specifically testing CBD oil for nightmare reduction. The evidence base is real but extrapolated — and understanding where it comes from helps you calibrate expectations appropriately.
CBD and Anxiety/PTSD: The Foundation
The most cited study in this space is Blessing et al. (2015), a systematic review finding broad preclinical and limited clinical evidence for CBD's anxiolytic effects across multiple anxiety disorder models — including PTSD.[1] Shannon et al. (2019) documented significant anxiety and sleep score improvements in a real-world psychiatric cohort using CBD; sleep scores improved in 66% of patients in the first month, though results fluctuated thereafter.[2] These are anxiety and sleep outcomes, not nightmare-specific outcomes — but given that PTSD nightmares are a sleep-disrupting anxiety disorder symptom, the translation is mechanistically reasonable.
The ECS and Fear Memory: Preclinical Evidence
Russo (2016) provides relevant mechanistic context in reviewing the ECS's role in stress, mood, and sleep — establishing that CB1 signaling in limbic structures is central to emotional regulation.[3] Crippa et al. (2011) demonstrated CBD's anxiolytic effects through neuroimaging, showing reduced amygdala activity and altered limbic-paralimbic connectivity with CBD versus placebo in a social anxiety disorder model.[4] This amygdala-dampening effect is directly relevant to the hyperactivated amygdala that drives trauma nightmares during REM.
Nabilone and PTSD Nightmares: The Closest Evidence
The most direct evidence for cannabinoids reducing PTSD nightmares involves nabilone — a synthetic THC analog, not CBD. Nabilone showed significant nightmare reduction in military veterans with PTSD in a small RCT, likely through CB1 agonism in the amygdala and NE modulation. This evidence doesn't transfer directly to CBD (which has no direct CB1 agonist activity), but it establishes that the ECS is a valid target for nightmare reduction in PTSD — and that targeting it pharmacologically can work.
Real-World CBD Use for Sleep
Corroon & Phillips (2018) surveyed over 2,400 CBD users and found sleep was among the top three reported uses, with high reported satisfaction — though nightmare reduction specifically was not isolated.[5] Capano et al. (2020) found significant improvements in sleep quality metrics in a clinical population using full-spectrum CBD — relevant context for understanding how CBD affects the sleep environment in which nightmares occur.[6]
For people with insomnia alongside nightmares, the sleep architecture benefits of CBD may be particularly valuable — improving sleep onset, reducing fragmentation, and supporting the restoration of normal sleep cycles that nightmares disrupt.
Image Rehearsal Therapy: The Gold Standard You Should Know About
Image Rehearsal Therapy (IRT) is the most evidence-supported non-pharmacological treatment for nightmare disorder and PTSD nightmares. The approach is straightforward: the patient writes out the recurring nightmare in detail, creates a new ending for the dream (one that is non-threatening or even positive), and rehearses this new version mentally while awake — typically for 10–20 minutes daily.
Over time, the brain incorporates the revised narrative into REM processing, reducing both the frequency and distress of the original nightmare. Multiple RCTs support IRT's effectiveness; current clinical guidelines for PTSD nightmares recommend IRT as a first-line treatment, often combined with prazosin.
CBD oil cannot replace IRT for nightmare disorder. However, for some people, nightmares are so intense and anxiety is so high that engaging with the nightmare content in IRT feels impossible. CBD's anxiolytic effects may reduce this barrier — making nightmare content less destabilizing to work with therapeutically. Think of it as a potential adjunct that might make evidence-based treatment more accessible, not a substitute for it.
CBD Oil vs. Prazosin for Nightmares
Prazosin is the most evidence-supported pharmacological treatment for PTSD nightmares. As an alpha-1 adrenergic receptor blocker, it directly targets the noradrenergic hyperactivity that drives trauma nightmares during REM — blocking NE signaling at the synapse. Multiple RCTs support its effectiveness, and it's included in current PTSD treatment guidelines specifically for nightmare reduction.
CBD's mechanism is indirect by comparison. Rather than blocking NE receptors directly, CBD modulates the HPA axis and ECS to reduce overall noradrenergic tone — a more diffuse effect with less predictable nightmare-specific outcomes. CBD also hasn't been tested in the direct comparative RCT format that would allow a meaningful evidence-level comparison with prazosin.
This doesn't mean CBD is without value — it means its evidence profile is different. CBD may be appropriate for people who find prazosin's side effects (notably orthostatic hypotension, dizziness) prohibitive, for those with stress-related nightmares that don't meet the threshold for PTSD treatment, or as an adjunct alongside established therapies. Important: if you're already taking prazosin and considering adding CBD oil, see the drug interactions section below — there's an additive hypotensive effect that requires medical supervision.
Why Broad Spectrum CBD Oil — Not Full Spectrum — for Nightmares
This distinction matters more for nightmares than almost any other CBD application. THC — present in full-spectrum products — has a complex and often counterproductive relationship with sleep and dreams. At low doses, THC can initially reduce REM sleep and suppress nightmares. But over time, and particularly with regular use, THC can increase anxiety, cause paranoia, and — critically — many users report intensified and more disturbing dream content with regular THC use, especially during periods of tolerance breaks when REM rebound occurs.
For people dealing with PTSD nightmares where the content is already intensely distressing, adding THC introduces unpredictability. Broad spectrum CBD oil delivers the full range of minor cannabinoids and terpenes (the entourage effect) without the THC that can worsen anxiety-driven dream content. Our Broad Spectrum CBD Oil is formulated for exactly this — comprehensive cannabinoid support with zero detectable THC.
Product Guide: Choosing the Right CBD Oil Format for Nightmares
| Product | Best For | Timing | Notes |
|---|---|---|---|
| Broad Spectrum CBD Oil | Pre-sleep anxiety, flexible dosing | 30–45 min before bed | Fast sublingual onset; zero THC; most adjustable format |
| CBD+CBN+Melatonin Gummies | Sleep maintenance + nightmare reduction | 60–90 min before bed | CBN adds sedative synergy; melatonin supports circadian regulation |
| Broad Spectrum Softgels | Daily ECS baseline, HPA axis support | Any time (consistent daily use) | Consistent systemic delivery; best for long-term HPA/ECS tone |
| Broad Spectrum Gummies | Flexible evening dose, convenience | 1–2 hours before bed | Longer onset, sustained release through early sleep |
| Full Spectrum Berry Gummies | Entourage effect (non-nightmare users) | Evening | Contains trace THC — use with caution for nightmare reduction; may be less predictable |
For nightmare-specific concerns, the CBD+CBN+Melatonin Gummies and Broad Spectrum Oil are typically the starting point. Adding daily Softgels for consistent baseline ECS support can complement evening dosing, particularly for people dealing with chronic stress or PTSD where daytime HPA dysregulation contributes to nighttime symptom burden.
Also explore our Elderberry Broad Spectrum Gummies for a broad-spectrum option with added immune support — useful during periods of high stress when nightmare frequency often increases alongside compromised immune function.
Drug Interactions: Nightmare-Specific Medications
If you're managing PTSD nightmares with prescription medications, CBD oil's potential drug interactions deserve careful attention. CBD is metabolized via CYP450 enzymes (primarily CYP2C19, CYP3A4, CYP2D6) and can inhibit or compete with medications processed by these pathways. For a comprehensive overview, see our guide on CBD oil drug interactions.
Drug Interaction Alert
Prazosin (PTSD nightmare standard): Alpha-1 blocker. CBD + prazosin can produce additive blood pressure-lowering effects, increasing the risk of orthostatic hypotension (dizziness when standing). Risk level: MODERATE. Medical supervision required before combining.
SSRIs (sertraline, paroxetine — PTSD first-line treatment): CYP2D6/CYP2C19 interactions possible. CBD may alter SSRI plasma levels. Risk: MODERATE. Monitor with prescriber.
Venlafaxine (PTSD): CYP2D6 substrate. Similar interaction profile to SSRIs. Risk: MODERATE.
Antipsychotics (quetiapine — used off-label for PTSD nightmares): CYP3A4 substrate. CBD may increase quetiapine levels. Risk: MODERATE. Discuss with prescriber.
Alcohol: Alcohol suppresses REM initially and then causes REM rebound — intensifying dreams and nightmares on the backend. CBD does not produce this rebound effect and may actually reduce alcohol-driven sleep disruption, but combining CBD with alcohol can increase sedation. Avoid alcohol if nightmare management is a priority.
Frequently Asked Questions
Does CBD oil help with nightmares?
CBD oil may help reduce nightmare frequency and intensity through several converging mechanisms — primarily by reducing pre-sleep anxiety and hyperarousal, modulating amygdala reactivity via 5-HT1A partial agonism, supporting endocannabinoid-mediated fear extinction during REM sleep, and dampening HPA axis activation that drives nocturnal noradrenergic hyperactivity. At moderate doses, CBD may also slightly reduce REM duration, which reduces nightmare opportunities without substantially impairing emotional processing. The evidence is promising but largely extrapolated from anxiety and PTSD research rather than nightmare-specific trials. Results vary by nightmare type, underlying cause, and individual ECS responsiveness.
Does CBD oil help with PTSD nightmares specifically?
PTSD-associated nightmares are the nightmare type with the most mechanistically plausible case for CBD oil. The ECS is embedded in the same neural circuits that dysregulate in PTSD — the amygdala, the vmPFC, and the HPA axis. CBD's capacity to reduce amygdala hyperreactivity, enhance fear extinction consolidation during REM, and lower the noradrenergic tone that drives trauma nightmares is theoretically strong. Human trial evidence is limited but directionally supportive — Shannon et al. (2019) documented significant sleep improvements in anxiety/PTSD populations using CBD. For a full discussion of CBD in PTSD management, see our CBD oil for PTSD post.
How does CBD oil affect REM sleep and dreaming?
CBD's effects on REM sleep are dose-dependent. At low doses (15–25mg), REM architecture is largely unchanged — CBD works primarily through anxiety reduction and ECS modulation rather than direct sleep architecture alteration. At moderate doses (25–60mg), studies suggest a mild reduction in REM duration. At higher doses (60mg+), more significant REM suppression occurs, potentially reducing dream frequency and intensity but also reducing the emotional processing and memory consolidation that REM provides. For nightmare management, moderate dosing tends to produce the best balance — some reduction in nightmare frequency without wholesale suppression of the REM sleep the brain needs for resilience. For a broader overview of CBD's effects on sleep architecture, see our guide to CBD oil for sleep.
Can CBD oil cause nightmares?
CBD oil itself is not associated with causing or worsening nightmares. If anything, CBD's anxiolytic and amygdala-modulating effects would be expected to reduce nightmare intensity, not increase it. However, this is another reason why broad spectrum formulations are strongly preferred over full-spectrum (THC-containing) products for nightmare management. THC has a complex relationship with dreams: it initially suppresses REM, which can reduce nightmares short-term, but regular THC use can increase anxiety and paranoia in some individuals — potentially worsening nightmare content. Additionally, during THC tolerance breaks, REM rebound can produce intensified, highly vivid dreams. CBD without THC avoids this complication entirely.
What dose of CBD oil is best for nightmares?
There is no established optimal dose for nightmares specifically. Based on the anxiety, PTSD, and sleep literature, a moderate dose range of 25–60mg taken 30–90 minutes before bed is a reasonable starting point. Lower doses (15–25mg) may be sufficient for stress-related nightmares where the primary mechanism is anxiety reduction. Higher doses may produce more REM suppression — reducing nightmare frequency but also potentially impairing emotional processing during sleep, which is counterproductive over the long term for PTSD. The standard approach is to start at the lower end, assess after 2–4 weeks, and adjust incrementally. See our CBD oil dosage guide for a detailed framework.
Is CBD oil safe to take with prazosin for PTSD nightmares?
This combination requires medical supervision. Both CBD and prazosin can lower blood pressure — CBD through HPA axis modulation and vascular effects, prazosin through direct alpha-1 adrenergic blockade. The combination may produce additive hypotensive effects, increasing the risk of orthostatic hypotension (dizziness upon standing, lightheadedness, fainting risk). This doesn't mean the combination is impossible — it means it should be initiated under medical guidance with monitoring of blood pressure. If you're currently taking prazosin for PTSD nightmares and want to explore adding CBD oil, discuss the plan with your prescribing physician before starting. For more on this topic, see our post on CBD oil drug interactions.
Why do I have more vivid dreams when I stop taking CBD oil?
This is a REM rebound phenomenon. If you've been using CBD at doses that modestly suppress REM sleep, stopping CBD — especially abruptly — can produce a compensatory surge in REM sleep duration and intensity in the nights that follow. This produces more vivid, memorable, and potentially more emotionally intense dreams. This is similar (though generally milder) to the REM rebound seen with alcohol cessation, benzodiazepine tapering, or REM-suppressing medication discontinuation. The effect is typically temporary — usually 3–7 days — and resolves as sleep architecture normalizes. To minimize it, taper CBD dose gradually rather than stopping cold, particularly if you've been using higher doses consistently for several weeks or more. This is also relevant context for understanding why dream vividness seems to increase the first few nights after stopping CBD.
Does CBD oil work better than prazosin for nightmares?
No — based on current evidence, prazosin is the more evidence-supported pharmacological option for PTSD nightmares specifically. Prazosin directly blocks the noradrenergic hyperactivity that drives trauma nightmares during REM, and its effectiveness has been demonstrated in multiple controlled trials with nightmare-specific endpoints. CBD oil's mechanisms are indirect by comparison, and there are no direct RCTs comparing the two for nightmare outcomes. CBD may have advantages in tolerability profile (prazosin commonly causes orthostatic hypotension and dizziness), may be more accessible, and may be more relevant for non-PTSD nightmare presentations. But anyone managing PTSD nightmares should be aware that current clinical guidelines recommend prazosin and IRT as primary interventions — CBD remains an adjunct at this point in the evidence base.
Can CBD oil help with nightmares that aren't related to trauma?
Yes — CBD oil may help with stress-related and idiopathic nightmares, though the evidence is even more limited than for PTSD nightmares. For stress-related nightmares, CBD's anxiolytic and HPA axis-modulating effects address the primary driver directly. Reducing pre-sleep anxiety, lowering evening cortisol, and dampening the stress response that carries into sleep can all reduce nightmare frequency. For idiopathic nightmares with no clear cause, improving overall sleep quality and reducing sleep fragmentation (which CBD may support) reduces nightmare opportunities, since nightmares most commonly occur in later REM cycles. Consider pairing CBD with good sleep hygiene — consistent sleep/wake times, limited blue light before bed, alcohol avoidance — to address nightmares from multiple angles. See our guide on CBD oil for stress for the anxiety mechanism in detail.
How long does CBD oil take to reduce nightmare frequency?
This varies significantly based on the mechanism at play. For pre-sleep anxiety reduction (the most immediate pathway), CBD oil can produce noticeable effects within the first few nights of use, though individual response varies. For sustained changes in nightmare frequency driven by ECS tone, HPA axis modulation, and fear extinction support, 4–8 weeks of consistent use is a more realistic timeframe for meaningful assessment. Research on CBD for anxiety and sleep suggests that sleep outcomes often improve within 1–2 months of consistent use, with individual variability. For PTSD-associated nightmares specifically, where the underlying fear memory networks are deeply entrenched, the timeline may be longer — and therapeutic approaches like IRT should be pursued alongside CBD rather than waiting to see if CBD alone produces full resolution. Tracking nightmare frequency in a sleep journal during the first 6–8 weeks of CBD use helps identify whether it's working and at what dose.
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References
- Blessing EM, Steenkamp MM, Manzanares J, Marmar CR. Cannabidiol as a Potential Treatment for Anxiety Disorders. Neurotherapeutics. 2015;12(4):825–836. PMID: 26341731
- Shannon S, Lewis N, Lee H, Hughes S. Cannabidiol in Anxiety and Sleep: A Large Case Series. Perm J. 2019;23:18–041. PMID: 30624194
- Russo EB. Clinical Endocannabinoid Deficiency Reconsidered: Current Research Supports the Theory in Migraine, Fibromyalgia, Irritable Bowel, and Other Treatment-Resistant Syndromes. Cannabis Cannabinoid Res. 2016;1(1):154–165. PMID: 28861491
- Crippa JA, Derenusson GN, Ferrari TB, et al. Neural basis of anxiolytic effects of cannabidiol (CBD) in generalized social anxiety disorder: a preliminary report. J Psychopharmacol. 2011;25(1):121–130. PMID: 20829306
- Corroon J, Phillips JA. A Cross-Sectional Study of Cannabidiol Users. Cannabis Cannabinoid Res. 2018;3(1):152–161. PMID: 30014049
- Capano A, Weaver R, Burkman E. Evaluation of the effects of CBD hemp extract on opioid use and quality of life indicators in chronic pain patients. Postgrad Med. 2020;132(1):56–61. PMID: 31711352
Medical Disclaimer
This article is for informational purposes only and does not constitute medical advice. CBD products have not been evaluated by the FDA and are not intended to diagnose, treat, cure, or prevent any disease or condition, including nightmare disorder or PTSD. If you experience chronic nightmares, especially related to trauma, please consult a qualified mental health professional. Do not discontinue prescribed medications without medical supervision. Individual results vary.
